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4. Small, Dense LDL. Some have argued, as I mentioned above, that only small, dense LDL contributes to arteriosclerosis. In fairness, they argue this because small, dense LDL has some unique properties:
And the research does indeed show that this kind of LDL is particularly viscious.  A number of studies have shown this clearly: "Particularly atherogenic forms of LDL include small, dense LDL particles and oxidized LDL. All lipoproteins that contain apolipoprotein B, such as LDL, very-low-density lipoprotein, and intermediate-density lipoprotein, tend to promote atherosclerosis." 
5. All Arterial Plaque Reversers. There are a number of well-known doctors out there who are actually reversing arteriosclerosis. This is a remarkable accomplishment if you think about it, because heart disease is the number one killer of men. Every one of these clinicians includes in their practice a strong LDL-lowering in strategy. This includes the famous physicians Drs. Davis, Esselstyn and Gould for example. Each of them has their own protocol for lowering LDL, but all of them have a proven track record of reducing arteriosclerosis by starting with LDL as their base technique.
And what LDL levels do they want? Is LDL of 120+ like the typical American okay? Each of them wants LDL to be in the 60-85 range, something I document in my link on LDL Thresholds for Arterial Health. By the way, the books of these authors are a fascinating read and not all of them are traditional Low Fat. One rule that I really like Dr. Davis' "Rule of 60" for plaque reversal: LDL < 60; HDL > 60; Triglycerides < 60.
6. Primal Cultures. Loren Cordain documented, in one of his early papers, how every modern primal culture with no heart disease had cholesterol below 150.  Of course, Loren Cordain is the founder and cheif apostle of the Paleo Diet, and many Paleo followers will be shocked to learn that Loren Cordain originally advocated LDL in the 50-70 range, because of the overwhelming eviden'ce of good health from these supposedly "primitive" peoples that were so heart healthy. 
Now cholesterol is not that relevant of a number, but cholesterol of 150 will almost always mean low LDL. If your cholesterol is low, then your LDL is very likely to be low as well and this is why Loren Cordain came to his original conclusions. Even the Masai, who ate boatloads of saturated fat had cholesterol right at 150. Some people say the Eskimo/Intuits had cholesterol greater than 150, but they also ate mountains of fish and suffered with severe osteoporosis-related issues because of their diet.
Again, do not fall for the idea that your LDL number does not matter and that it is only inflammation and triclycerides that count. Consider what these authors wrote: "Thus, large and small LDL are atherogenic, and it is not possible to judge which if any is more harmful, overall."  Does this mean that all men with medium or high levels of LDL will develop arteriosclerosis? None of us can say always of course. But it does mean that you are taking your life into your own hands.
So what can you do? It is simple. I have a link with good starter information called How to Clear Your Arteries and I also highly recommend that you read the books of the above doctors. Particularly relevant is Prevent and Reverse Heart Disease by Dr. Esselstyn and Track Your Plaque by Dr. Davis , both of whom generally advocate a drug-free approach.
CAUTION: If you have a medical condition or are on any medications, please discuss any changes with your doctor first. Certain supplements, foods and even juices can alter absorption rates of certain medications for example. Play it safe.
1) http://thepaleodiet.com/wp-content/uploads/2012/11/JACC-LDL-Final.pdf, JACC, June 2, 2004:2142–62004, 43(11), "Optimal Low-Density Lipoprotein Is 50 to 70 mg/dl: Lower Is Better and Physiologically Normal"
2) The Journal of Clinical Endocrinology & Metabolism, Oct 1 2003, 88(10), "Low-Density Lipoprotein Size and Cardiovascular Disease: A Reappraisal"
3) Circulation, 2004, 109:III-2-III-7, "Atherosclerosis: Evolving Vascular Biology and Clinical Implications: Atherogenic Lipoprotein Particles in Atherosclerosis"
4) Can J Cardiol, 2001 Aug, 17(8):859-65, "A prospective, population-based study of low density lipoprotein particle size as a risk factor for ischemic heart disease in men"
5) Arteriosclerosis, Thrombosis, and Vascular Biology, 1998, 18:577-583, "IDL Composition and Angiographically Determined Progression of Atherosclerotic Lesions During Simvastatin Therapy"
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